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Caldikind (Calcitriol)

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Caldikind is a hormonally-active, synthetic vitamin D analog prescribed to treat hypocalcemia, osteoporosis, and the prevention of corticosteroid-induced osteoporosis. Systemic Caldikind is FDA indicated to control hypocalcemia in patients on chronic renal dialysis, secondary hyperparathyroidism in those with chronic kidney disease not yet on dialysis, and hypocalcemia in patients with hypoparathyroidism and pseudohypoparathyroidism.

Other names for this medication:
Alfabond, Alfacal, Alfacalcirol, Alfacip, Arachitol, Arcium, Aristrol, Biocal, Bonetuf, Calbec, Calbory, Calbovic, Calcicare, Calcirol, Calday, Caldix, Calfa, Calgel, Calsure, Caltin, Caltop, Calzem, Carcal, Denscal, Devita, Ikmag, Lctrol, Minroset, Netxod, Novadee, Ostriol, Politrol, Primacal, Qpcal, Rical, Riecal, Ritoca, Rockbon, Roical, Rolsical, Septriol, Tobalt, Vezocal, Vitalpha

Similar Products:
Trileptal, Ovral, Eulexin, Lodine, Feldene, Levaquin, Indocin, Atarax, Flonase

Also known as: Calcitriol


Generic Caldikind is a synthetic vitamin D analog which is active in the regulation of the absorption of calcium from the gastrointestinal tract and its utilization in the body. The two known sites of action of Generic Caldikind are intestine and bone. Generic Caldikind receptor-binding protein appears to exist in the mucosa of human intestine. Additional evidence suggests that Generic Caldikind may also act on the kidney and the parathyroid glands. Generic Caldikind is the most active known form of vitamin D3 in stimulating intestinal calcium transport. In acutely uremic rats Generic Caldikind has been shown to stimulate intestinal calcium absorption. The kidneys of uremic patients cannot adequately synthesize Generic Caldikind, the active hormone formed from precursor vitamin D. Resultant hypocalcemia and secondary hyperparathyroidism are a major cause of the metabolic bone disease of renal failure. However, other bone-toxic substances which accumulate in uremia (eg, aluminum) may also contribute. The beneficial effect of Generic Caldikind in renal osteodystrophy appears to result from correction of hypocalcemia and secondary hyperparathyroidism. It is uncertain whether Generic Caldikind produces other independent beneficial effects. Caldikind is also known as Calcitriol. Generic Caldikind treatment is not associated with an accelerated rate of renal function deterioration. No radiographic evidence of extraskeletal calcification has been found in predialysis patients following treatment. The duration of pharmacologic activity of a single dose of calcitriol is about 3 to 5 days. Generic name of Generic Caldikind is Calcitriol. Brand name of Generic Caldikind is Caldikind.


Generic Caldikind is available in capsules (0.25 mcg) and liquid form. Capsules must be swallowed whole. Do not crush or chew them. If you are prescribed the liquid form, use as directed. Follow dosing instructions closely. Do not take this medication with mineral oil or magnesium containing antacids which may reduce its effects. Foods rich in vitamin D include: fortified dairy products, eggs, sardines, cod liver oil, chicken livers and fresh-water fish. Vitamin D is also obtained from periodic exposure to the sun. It is important to maintain a well-balanced diet while taking this medication. Calcium supplements may be recommended in addition to this medication. If you want to achieve most effective results do not stop taking Generic Caldikind suddenly.


If you overdose Generic Caldikind and you don't feel good you should visit your doctor or health care provider immediately.


Store at room temperature between 15 and 30 degrees C (59 and 86 degrees F) away from moisture, light and heat. Keep container tightly closed. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

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The most common side effects associated with Caldikind are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.


Problems in children have not been reported with intake of normal daily recommended amounts. Some studies have shown that infants who are totally breast-fed, especially with dark-skinned mothers, and have little exposure to sunlight may be at risk of vitamin D deficiency. Your health care professional may prescribe a vitamin/mineral supplement that contains vitamin D. Some infants may be sensitive to even small amounts of alfacalcidol, calcifediol, calcitriol, dihydrotachysterol, or ergocalciferol. Also, children may show slowed growth when receiving large doses of alfacalcidol, calcifediol, calcitriol, dihydrotachysterol, or ergocalciferol for a long time.

Studies on doxercalciferol or paricalcitol have been done only in adult patients, and there is no specific information comparing the use of doxercalciferol or paricalcitol in children with use in other age groups.

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Special attention is given to the sporadic character of the present cases in Roumania with emphasis on the seasonal repartition of cases, occupation of subjects, and urban versus rural proportion of cases. Our calculations show that an accuracy of 10-4 to 10-7 Eh can be reached in HF-SCF calculations on general molecules. We performed a retrospective study to assess whether interview timing was associated with successful matching at our IM program. There are several user-based and expert-based usability evaluation methods that may perform differently according to the context in which they are used. The rapid, highly sensitive and specific measurement of BNP concentrations in the patients with dyspnea can significantly help to differentiate the cardiac and pulmonary causes of dyspnea (Tab. Deubiquitinase Usp18 prevents cellular apoptosis from oxidative stress in liver cells. Its incidence and mode of presentation in Kenyan Africans are unknown. The role and impact of research positions within health care settings in allied health: a systematic review. The results indicate that a number of mechanisms may be involved in the deleterious effects of chromate on male fecundity.

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Kinetic and mechanistic studies of vanadium-based, extended catalytic lifetime catechol dioxygenases. We tested this parasympathetic rebound hypothesis by presenting animals with a fear-inducing (sympathetic-activating) conditioned stimulus (CS) after 2 hr of water-restraint stress. 4-HNE is an important lipid peroxidation product which has been implicated in the mutagenesis and carcinogenesis processes. A discriminative dictionary representation method was developed to mitigate CT truncation artifacts directly in the DICOM image domain. Protein kinases of the Raf family phosphorylate and activate the mitogen-activated protein kinase kinases (MKKs, also known as MEKs). It has been shown that COX-2 inhibitors have inhibitory activities against various types of tumor, including colorectal carcinoma. The DCIA offers good quality and quantity of bone tissue for mandibular segmental defect and implant for dental rehabilitation. Interruption of the cerebellum red nuclear path at the medial part of midbrain was considered to be the reason for bilateral cerebellar ataxia and visualization of cerebellum red nuclear path by DTI can give better understanding of the neurological symptom. This new cell line was established what is the recommended dose of effexor directly from a primary woodchuck hepatocellular carcinoma.

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