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Atedine (Atenolol)

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Atedine belongs to a class of drugs known as beta blockers. It works by blocking the action of certain natural chemicals in your body, such as epinephrine, on the heart and blood vessels. Atedine is used alone or in combination with other medications.

Other names for this medication:
Aloten, Amlica, Amloact, Amlocal, Amlocom, Amlokind, Amlonol, Amloten, Amlotest, Amlotin, Ampilife, Andinx, Apine, Atcom, Atzee, Cyplol, Etotan, Glipin, Harten, Licopin, Lonet, Melol, Myodura, Nusar, Olate, Pdnol, Pericard, Presolar, Primodil, Stressban, Teenol, Tenlol, Tenochek, Tensimin, Tentoric, Vascodil

Similar Products:
Uroxatral, Ceclor, Sumycin, Diovan, Urispas

Also known as: Atenolol


Tenormin (atenolol) is a beta-blocker that affects the heart and circulation (blood flow through arteries and veins).

Tenormin is used to treat angina (chest pain) and hypertension (high blood pressure).

Tenormin is also used to lower the risk of death after a heart attack.


A nurse or other trained health professional will give you this medicine. This medicine is given through a needle placed in one of your veins .

Detailed Tenormin dosage information


Seek emergency medical attention or call the Poison Help line at 1-800-222-1222.

Overdose symptoms may include extreme weakness or lack of energy, very slow heart rate, shortness of breath, or fainting.


Store at room temperature between 20 and 25 degrees C (68 and 77 degrees F) away from moisture and heat. Keep container tightly closed. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Atedine are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.


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New Functions and Subcellular Localization Patterns of c-di-GMP Components (GGDEF Domain Proteins) in B. Isolation and characterization of a human gene that encodes a new subclass of protein tyrosine kinases. In vivo assessment of trabecular bone microarchitecture by high-resolution peripheral quantitative computed tomography. Behavioral tests were conducted on wild ambien dosage males -caught sires and their laboratory-reared sons. In differential diagnosis, conditions mimicking myopathy can be excluded on the basis of physical findings, various tests, and when appropriate, muscle biopsy. Stimulation by native HBsAg particles, which corresponded to the appearance of anti-HBs antibody, preceded that observed using synthetic peptides. Animal experiments suggest that neurochemical and anatomic asymmetries exist within the basal ganglia, particularly the globus pallidus, and that these asymmetries correlate with both preferred direction of rotation and limb preference in lever pressing. RNA was extracted from livers of 5 rats with hereditary analbuminemia (the Nagase analbuminemic rat, NAR), 5 rats with NS induced by adriamycin (Adria), 5 rats with NS caused by passive Heymann nephritis (NS) and 5 control animals. Use of different prostaglandin analogues for terminating pregnancy at different terms. Ten patients had clinical evidence of prostatitis and elevated PSA levels.

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Behavioral Problems and Socioemotional Competence at 18 to 22 Months of Extremely Premature Children. A subset of subjects (11 FHP, 11 FHN) was admitted to the General Clinical Research Center to measure serum cortisol levels every 30 min for 24 hr. Quantitative determination of multi markers in five varieties of Withania somnifera using ultra-high performance liquid chromatography with hybrid triple quadrupole linear ion trap mass spectrometer combined with multivariate analysis: Application to pharmaceutical dosage forms. prilosec dose Interestingly, the Ffar4 agonism-mediated signaling axis of Ffar4-miR-30b/378-Ucp1 was linked with an elevation of cAMP in brown adipocytes, similar to cold-exposed or fish oil-fed brown fat. In 9 patients the pacing rate could be compared with the underlying sinus rate during exercise and was seen to match it very closely. PCNA message levels were low at G0, gradually increased following entrance into G1, peaked at G1/S, and declined during S phase.

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